In most cases, water callus treatment can be done independently. Small calluses are usually not painful and do not break open on their own. Therefore, they are simply covered with a plaster that protects them from injury. If the water callus is large, it causes significant discomfort and is fraught with rupture. It is better to pierce such a “dropsy”. The most favorable time for puncture is considered to be the first day after the bubble appears on the skin.
  Piercing a water callus must be carried out with the obligatory observance of certain rules to avoid infection in the callus. First, it is necessary to disinfect the puncture site. To do this, lubricate the water callus with iodine or brilliant green. Secondly, the puncture can only be made with a sterile needle. You can take a regular pin or sewing needle. To sterilize it, you need to hold it in alcohol or heat it over a fire.
  The puncture should be made on the side of the water callus, inserting the needle almost parallel to the surface of the skin. Under no circumstances should you pierce the callus perpendicular to its surface, as this will cause the needle to injure the bottom of the callus. To ensure that the fluid outflow from the water callus occurs continuously, it is better to make several punctures. The main thing is not to overdo it and preserve the walls of the bubble. Like a natural bandage, they cover the delicate skin located inside the water callus and protect it from damage and infection.
  After the punctures are made, the water callus is gently pressed with a gauze cloth so that all the existing liquid comes out of it. If over time the callus becomes filled with contents again, it will be necessary to repeat the puncture. To prevent the development of infection after a puncture, an ointment containing an antibiotic is applied to the water callus. Then cover the callus with a protective plaster, which should be changed at least 2 times a day and removed at night.
  If the opening of the water callus occurs spontaneously and with a tearing of its wall, the resulting wound must be cleaned and disinfected, apply antibacterial ointment and cover the callus with a gauze pad and adhesive tape. The latter will protect the water callus from contamination and injury, creating favorable conditions for its healing.
  If an infection gets into a water callus, it is necessary to open it and remove the walls, since in a closed callus favorable conditions are created for the development of the infectious process and suppuration. Therefore, if infected, you should seek help from a surgeon. In compliance with all the rules of asepsis, he will perform an autopsy and initial treatment of the infected water callus, apply a bandage to ensure drainage of the wound and prescribe local antibacterial treatment.

Your feet definitely need special care, as plantar calluses can occur due to various factors. It is imperative to periodically arrange caring cosmetic procedures for your feet.

Calluses and corns often form on the toes and heels. This mainly happens due to wearing low-quality and tight shoes. You definitely need to pay attention to this problem and try to get rid of the existing problem as quickly as possible.

Types of calluses

Plantar callus (ICD-10 code L84) refers to neoplasms on the skin. Despite its unpleasant appearance, a callus performs a rather important function - it protects the skin from friction. They are often formed due to wearing tight shoes. Calluses are:

  • dry;
  • wet;
  • blood;
  • rod;
  • corns.

Dry calluses form in areas of high pressure, often on the feet. This is a layer of cells that protects the skin from constant injury. are formed when the skin is rubbed. Fluid begins to accumulate under the skin.

Blood calluses are somewhat similar to wet calluses, but instead of fluid, blood forms inside them. This occurs due to the close proximity of the vessels.

The callus gets its name due to its specific structure. It is a rough area of ​​skin protruding above its surface with a depression in the center. Inside the recess there is a rod or root that penetrates very deeply into the tissue. The size of the callus can be very different, it all depends on the degree of neglect of the disease. Their appearance on the feet is mainly due to the pressure of shoes.

Corns are similar in appearance to dry calluses, but they are more superficial. They appear with constant pressure on the skin and are mainly observed in runners and athletes. They can also be found in women who prefer to wear high heels.

Dry calluses

Externally, a dry plantar callus on the feet is very easy to diagnose, since it has a roughened structure. It protrudes above the surface of the skin and usually looks like a round yellowish spot. There is often some pain when pressed, but sometimes they are painless.

Due to constant pressure, the skin begins to thicken and increase in thickness, but it grows not outward, but inward. Dry calluses can appear on the heels, soles, and toes. Most often they form on the protruding parts of the foot.

Wet calluses

As a result of prolonged pressure on a certain area of ​​the skin, redness initially occurs, and then a bubble forms, inside which fluid accumulates. It is this that protects damaged skin and promotes faster healing. If the vessels in the affected area are located close to the surface, and the pressure is strong, then the fluid inside the callus may contain blood impurities.

A similar problem occurs as a result of wearing shoes that are too tight and do not fit properly. Blisters are caused by shoes that have too rough seams or are made of poor quality material. Wet plantar callus is characterized not only by pain. After the membrane of the skin surface ruptures, fluid leaks out and infection can enter the wound.

At the first sign of redness, be sure to change your shoes, then apply to the affected area. If appropriate measures are not taken in a timely manner, then in order to avoid complications, it is necessary to treat with surgery.

Callus

Many people believe that the callus is not dangerous at all and does not require any additional treatment. However, this is absolutely not true, since a core callus can form in place of a simple callus. It grows very deeply into the skin and causes pain and discomfort when moving, especially if it forms on the sole. In addition, a callus may form in the area of ​​the thumb.

The core plantar callus consists of two parts, namely the shaft and the cap. The root or rod penetrates very deeply into the tissue and holds onto it, making it impossible to remove the growth in the usual way. The cap is a bulge, in the center of which there is a small depression that has a rounded shape. Its size largely depends on the condition of the callus.

Corns

Quite often, hard plantar calluses or corns form, which mainly appear due to wearing tight or uncomfortable shoes. They are painful areas of keratinized, thickened skin. Corns can occur on the balls of your feet or your heels.

This type of plantar callus is quite common and can cause severe pain and burning while walking. However, pain and discomfort do not always occur, since corns generally do not cause pain or discomfort. In this case, they can only be detected by touch or visually. Many people are interested in how to distinguish a callus from a plantar wart. It is worth noting that corns do not have a core; they are more dense in structure.

Causes

One of the most common foot problems is plantar callus. It mainly occurs when wearing shoes that are too tight or poorly fitted. Among the main reasons for this problem are the following:

  • improper distribution of load when walking;
  • heel spur;
  • shoes that are not the right size;
  • bursitis;
  • flat feet;
  • vitamin deficiency;
  • joint arthritis;
  • osteoarthritis.

The main cause of plantar callus is increased stress on the foot, which is accompanied by constant pressure on the sole and rubbing. Among the main provoking factors it is necessary to highlight the following:

  • flat feet;
  • excess weight;
  • narrow or uncomfortable shoes.

However, increased physical stress on the foot only provokes and accelerates the process of skin roughening. In addition, among the causes of the formation of calluses on the sole of the foot, it is necessary to highlight various diseases of the internal organs.

What calluses can tell you

Plantar callus in a child and an adult can signal the presence of various diseases and disorders in the body. Based on their location, existing diseases can be diagnosed. If calluses have formed due to wearing uncomfortable shoes or having flat feet, they disappear immediately after changing them or choosing an arch support.

If calluses appear as a result of diseases in the body, then they can be eliminated only after treating disorders of this organ. Hard calluses along the edges of the heel may indicate the presence of pathological changes in the joints. Calluses that form on the outer area of ​​the foot indicate the presence of spinal diseases.

If a callus forms on the inside of the sole, this serves as a signal for an intestinal examination. Corns under the little finger indicate a problem with the liver or heart. The main sign of nervous overstrain in the body is a large callus on the sole opposite the toes. If you additionally experience a cough, this may be a sign of respiratory disease.

Thyroid dysfunction is indicated by rough skin along the edges of the big toes. If you are overweight, this sign may indicate a slow metabolism. In addition, corns can occur due to poor circulation in the legs, joint diseases and curvature of the spine.

Medications

Treatment of corns on the feet is carried out using various pharmaceutical products, which are presented in a wide variety. There are several options for such funds, namely:

  • plasters;
  • creams;
  • ointments;
  • liquids.

Each of these types of funds has its own specific pros and cons. Ointments for removing calluses and corns mainly contain salicylic acid. In addition, they often contain benzoic acid. Thanks to this, dry skin softens and is easily removed.

The ointment should be applied to dry, pre-steamed skin directly onto the callus. The patch is then attached. The ointment should be washed off after about 1-2 hours. When using such a product, you need to remember that the substances contained in such a product can cause irritation on healthy skin. That is why they must be used very carefully and only on affected areas of the skin. Among the ointments, “Super Antimozolin” and “Nemosol” should be highlighted.

Plasters are much more convenient than ointments, but their effectiveness is somewhat worse. The patch is applied to the affected area and lasts approximately 8 hours. After this, the callus becomes soft and can be removed very easily with a pumice stone or brush. If the required effect is not achieved, then the treatment must be repeated every other day. You can use such patches as “Luxplast”, “Entsy”, “Salipod”.

Treatment of corns on the feet can be carried out using liquid products. They need to be applied using a cloth. In particular, they use such products as “Lekker Stopmosol”, “Balsam Karavaeva Vitaon”.

Treatment with medications can be carried out only if the callus on the foot is not in advanced form. And in the presence of old growths, removal of plantar calluses is required, and for this purpose surgical methods are used, in particular, such as:

  • laser removal;
  • cryodestruction;
  • electrocoagulation;
  • radio wave surgery.

Cryodestruction means that with this method, dry callus is removed using liquid nitrogen. This substance has a very low temperature, due to which the tissue of the resulting growth is rejected. It is worth noting that the foot becomes clean and smooth.

Laser treatment is characterized by the fact that dead tissue is burned using a laser device. In addition, pathogenic microorganisms are destroyed in the area of ​​laser exposure. This method is considered the best and most effective. Moreover, it is non-traumatic.

Electrocoagulation means that dry calluses are exposed to electric current. This method is quite common. Radio wave surgery is used to remove warts, papillomas and many other skin growths. All these techniques are used when the legs are in poor condition.

You can also contact a cosmetologist who will help you quickly and effectively solve the problem. Almost all salons offer pedicure procedures. If the callus is not too old, then cosmetologists will help you quickly remove it.

If you have a callus with a core, your doctor may prescribe drilling to remove the callus with a core completely from the skin. At the same time, healthy areas are not affected, which contributes to their faster recovery. This is followed by long-term treatment aimed at eliminating inflammation and fungi. After the procedure, the doctor must make sure that the rod is completely removed.

Application of traditional methods

Many people are interested in how to get rid of plantar calluses using folk remedies and techniques. The treatment process will not be too fast, but this does not mean that the result will be worse or not at all. There are several different ways you can remove dry callus.

The most common treatment method is baths. To prepare them, you need to add soda and a little soap to the water. Stir everything well. Keep your feet in this product for 15 minutes. Then treat your feet with pumice.

Aloe is considered a good remedy. The plant contains many useful substances. The aloe leaf should be kept in a cold place for 3-4 days, then rubbed into the callus every day before bed.

You can mix aloe juice with fish oil and apply this mixture to the callus every day. This is best done before bed. In the morning, your feet should be washed and rinsed with chamomile decoction. This remedy must be used until the callus is completely removed.

To get rid of calluses, you can use propolis. The product is applied to the damaged area and fixed with adhesive tape. Relief will come after several procedures.

You can apply finely grated raw potatoes to the callus, then secure it well. Wrap your foot in compress paper and put on a sock. In the morning, rinse everything well. You can also use potato gruel along with onion gruel.

Carrying out prevention

To avoid the formation of calluses on your feet, you need to:

  • observe the rules of hygiene;
  • periodically remove the stratum corneum;
  • lubricate your feet with softening cream;
  • if you are prone to the formation of corns, consult an orthopedist;
  • if necessary, wear orthopedic insoles or shoes;
  • wear comfortable shoes.

In case of callus formation, it is important not to start this process, but to start treatment in a timely manner, as this will allow you to get rid of the problem much faster.

CLASS XII. DISEASES OF SKIN AND SUBcutaneous Fiber (L00-L99)

This class contains the following blocks:
L00-L04 Infections of the skin and subcutaneous tissue
L10-L14 Bullous disorders
L20-L30 Dermatitis and eczema
L40-L45 Papulosquamous disorders
L50-L54 Urticaria and erythema
L55-L59 Diseases of the skin and subcutaneous tissue associated with radiation
L60-L75 Skin appendage diseases
L80-L99 Other diseases of the skin and subcutaneous tissue

The following categories are marked with an asterisk:
L14*Bullous skin disorders in diseases classified elsewhere
L45* Papulosquamous disorders in diseases classified elsewhere

L54* Erythema in diseases classified elsewhere
L62* Changes in nails in diseases classified elsewhere
L86* Keratoderma in diseases classified elsewhere
L99* Other disorders of the skin and subcutaneous tissue in diseases classified elsewhere

INFECTIONS OF THE SKIN AND SUBCUTANEOUS FIBER (L00-L08)

If it is necessary to identify an infectious agent, use an additional code ( B95-B97).

Excluded: hordeolum ( H00.0)
infectious dermatitis ( L30.3)
local skin infections classified in class I,
such as:
erysipelas ( A46)
erysipeloid ( A26. -)
herpetic viral infection ( B00. -)
anogenital ( A60. -)
molluscum contagiosum ( B08.1)
mycoses ( B35-B49)
pediculosis, acariasis and other infestations ( B85-B89)
viral warts ( B07)
panniculitis:
NOS ( M79.3)
lupus ( L93.2)
neck and back ( M54.0)
recurrent [Weber-Christian] ( M35.6)
fissure of the lip commissure [jamming] (due to):
NOS ( K13.0)
candidiasis ( B37. -)
riboflavin deficiency ( E53.0)
pyogenic granuloma ( L98.0)
herpes zoster ( B02. -)

L00 Staphylococcal skin lesion syndrome in the form of burn-like blisters

Pemphigus of the newborn
Ritter's disease
Excluded: toxic epidermal necrolysis [Lyella] ( L51.2)

L01 Impetigo

Excluded: impetigo herpetiformis ( L40.1)
pemphigus of the newborn ( L00)

L01.0 Impetigo [caused by any organism] [any location]. Impetigo Bockhart
L01.1 Impetiginization of other dermatoses

L02 Skin abscess, boil and carbuncle

Included: boil
furunculosis
Excluded: areas of the anus and rectum ( K61. -)
genital organs (external):
female ( N76.4)
men's ( N48.2, N49. -)

L02.0 Skin abscess, boil and carbuncle of the face
Excluded: external ear ( H60.0)
century ( H00.0)
head [any part other than the face] ( L02.8)
lacrimal:
glands ( H04.0)
paths ( H04.3)
mouth ( K12.2)
nose ( J34.0)
eye sockets ( H05.0)
submandibular ( K12.2)
L02.1 Skin abscess, boil and carbuncle of the neck

L02.2 Skin abscess, boil and carbuncle of the trunk. Abdominal wall. Back [any part other than the gluteal]. Chest wall. Groin area. Crotch. Navel
Excluded: breast ( N61)
pelvic girdle ( L02.4)
omphalitis of the newborn ( P38)
L02.3 Skin abscess, boil and carbuncle of the buttock. Gluteal region
Excluded: pilonidal cyst with abscess ( L05.0)
L02.4 Skin abscess, boil and carbuncle of the limb
L02.8 Skin abscess, boil and carbuncle of other localizations
L02.9 Skin abscess, boil and carbuncle of unspecified localization. Furunculosis NOS

L03 Phlegmon

Included: acute lymphangitis
Excluded: phlegmon:
areas of the anus and rectum ( K61. -)
external auditory canal ( H60.1)
external genitalia:
female ( N76.4)
men's ( N48.2, N49. -)
century ( H00.0)
lacrimal apparatus ( H04.3)
mouth ( K12.2)
nose ( J34.0)
eosinophilic cellulitis [Velsa] ( L98.3)
febrile (acute) neutrophilic dermatosis [Svita] ( L98.2)
lymphangitis (chronic) (subacute) ( I89.1)

L03.0 Phlegmon of the fingers and toes
Nail infection. Onychia. Paronychia. Peronychia
L03.1 Phlegmon of other parts of the extremities
Armpit. Pelvic girdle. Shoulder
L03.2 Facial phlegmon
L03.3 Phlegmon of the trunk. Abdominal walls. Back [any part]. Chest wall. Groin. Crotch. Navel
Excluded: omphalitis of the newborn ( P38)
L03.8 Phlegmon of other localizations
Head [any part other than the face]. Scalp
L03.9 Cellulitis, unspecified

L04 Acute lymphadenitis

Includes: abscess (acute) of any lymph node,
acute lymphadenitis) except mesenteric
Excluded: swollen lymph nodes ( R59. -)
disease caused by human immunodeficiency virus
[HIV], manifested as a generalized
lymphadenopathy ( B23.1)
lymphadenitis:
NOS ( I88.9)
chronic or subacute, except mesenteric ( I88.1)
mesenteric nonspecific ( I88.0)

L04.0 Acute lymphadenitis of the face, head and neck
L04.1 Acute lymphadenitis of the trunk
L04.2 Acute lymphadenitis of the upper limb. Armpit. Shoulder
L04.3 Acute lymphadenitis of the lower limb. Pelvic girdle
L04.8 Acute lymphadenitis of other localizations
L04.9 Acute lymphadenitis, unspecified

L05 Pilonidal cyst

Includes: fistula - coccygeal or
sinus) pilonidal

L05.0 Pilonidal cyst with abscess
L05.9 Pilonidal cyst without abscesses. Pilonidal cyst NOS

L08 Other local infections of the skin and subcutaneous tissue

L08.0 Pyoderma
Dermatitis:
purulent
septic
pyogenic
Excludes: pyoderma gangrenous ( L88)
L08.1 Erythrasma
L08.8 Other specified local infections of the skin and subcutaneous tissue
L08.9 Local infection of skin and subcutaneous tissue, unspecified

BULLOUS DISORDERS (L10-L14)

Excludes: benign (chronic) familial pemphigus
[Hailey-Hailey disease] ( Q82.8)
syndrome of staphylococcal skin lesions in the form of burn-like blisters ( L00)
toxic epidermal necrolysis [Lyell's syndrome] ( L51.2)

L10 Pemphigus [pemphigus]

Excludes: pemphigus of the newborn ( L00)

L10.0 Pemphigus vulgare
L10.1 Pemphigus vegetans
L10.2 Pemphigus foliaceus
L10.3 Brazilian bladderwort
L10.4 Pemphigus is erythematous. Senir-Usher syndrome
L10.5 Drug-induced pemphigus
L10.8 Other types of pemphigus
L10.9 Pemphigus, unspecified

L11 Other acantholytic disorders

L11.0 Acquired keratosis follicularis
Excluded: keratosis follicularis (congenital) [Darrieu-White] ( Q82.8)
L11.1 Transient acantholytic dermatosis [Grover's]
L11.8 Other specified acantholytic changes
L11.9 Acantholytic changes, unspecified

L12 Pemphigoid

Excluded: pregnancy herpes ( O26.4)
impetigo herpetiformis ( L40.1)

L12.0 Bullous pemphigoid
L12.1 Scarring pemphigoid. Benign pemphigoid of the mucous membranes [Levera]
L12.2 Chronic bullous disease in children. Juvenile dermatitis herpetiformis
L12.3 Epidermolysis bullosa acquired
Excludes: epidermolysis bullosa (congenital) ( Q81. -)
L12.8 Other pemphigiodes
L12.9 Pemphigoid, unspecified

L13 Other bullous changes

L13.0 Dermatitis herpetiformis. Dühring's disease
L13.1 Subcorneal pustular dermatitis. Sneddon-Wilkinson disease
L13.8 Other specified bullous changes
L13.9 Bullous changes, unspecified

L14* Bullous skin disorders in diseases classified elsewhere

DERMATITIS AND ECZEMA (L20-L30)

NoteIn this block, the terms “dermatitis” and “eczema” are used interchangeably as synonyms.
Excluded: chronic (childhood) granulomatous disease ( D71)
dermatitis:
dry skin ( L85.3)
artificial ( L98.1)
gangrenous ( L88)
herpetiformis ( L13.0)
perioral ( L71.0)
stagnant ( I83.1 I83.2 )
diseases of the skin and subcutaneous tissue associated with exposure to radiation ( L55-L59)

L20 Atopic dermatitis

Excluded: limited neurodermatitis ( L28.0)

L20.0 Scabies Beignets
L20.8 Other atopic dermatitis
Eczema:
flexion NEC
pediatric (acute) (chronic)
endogenous (allergic)
Neurodermatitis:
atopic (localized)
diffuse
L20.9 Atopic dermatitis, unspecified

L21 Seborrheic dermatitis

Excluded: infectious dermatitis ( L30.3)

L21.0 Seborrhea of ​​the head. "Baby's Cap"
L21.1 Seborrheic infantile dermatitis
L21.8 Other seborrheic dermatitis
L21.9 Seborrheic dermatitis, unspecified

L22 Diaper dermatitis

Diaper:
erythema
rash
Psoriasis-like diaper rash

L23 Allergic contact dermatitis

Included: allergic contact eczema
Excludes: allergy NOS ( T78.4)
dermatitis:
NOS ( L30.9)
contact BDU ( L25.9)
diaper ( L22)
L27. -)
century ( H01.1)
simple irritable contact ( L24. -)
perioral ( L71.0)
eczema of the outer ear ( H60.5)
diseases of the skin and subcutaneous tissue associated with exposure to radiation ( L55-L59)

L23.0 Allergic contact dermatitis caused by metals. Chrome. Nickel
L23.1 Allergic contact dermatitis caused by adhesives
L23.2 Allergic contact dermatitis caused by cosmetics
L23.3 Allergic contact dermatitis caused by drugs in contact with the skin
If it is necessary to identify the drug, use an additional code for external causes (class XX).
T88.7)
L27.0-L27.1)
L23.4 Allergic contact dermatitis caused by dyes
L23.5 Allergic contact dermatitis caused by other chemicals
With cement. Insecticides. Plastic. Rubber

L23.6 Allergic contact dermatitis caused by food contact with skin
L27.2)
L23.7 Allergic contact dermatitis caused by plants other than food
L23.8 Allergic contact dermatitis caused by other substances
L23.9 Allergic contact dermatitis, cause not specified. Allergic contact eczema NOS

L24 Simple irritant contact dermatitis

Included: simple irritant contact eczema
Excludes: allergy NOS ( T78.4)
dermatitis:
NOS ( L30.9)
allergic contact ( L23. -)
contact BDU ( L25.9)
diaper ( L22)
caused by substances taken orally ( L27. -)
century ( H01.1)
perioral ( L71.0)
eczema of the outer ear ( H60.5)
diseases of the skin and subcutaneous tissue associated
with exposure to radiation ( L55-L59)

L24.0 Simple irritant contact dermatitis caused by detergents
L24.1 Simple irritant contact dermatitis caused by oils and lubricants
L24.2 Simple irritant contact dermatitis caused by solvents
Solvents:
chlorine-containing)
cyclohexane)
ethereal)
glycolic) group
hydrocarbon)
ketone)
L24.3 Simple irritant contact dermatitis caused by cosmetics
L24.4 Irritant contact dermatitis caused by drugs in contact with the skin
If it is necessary to identify the drug, use an additional code for external causes (class XX).
Excludes: drug-induced allergy NOS ( T88.7)
drug-induced dermatitis ( L27.0-L27.1)
L24.5 Simple irritant contact dermatitis caused by other chemicals
With cement. Insecticides
L24.6 Simple irritant contact dermatitis caused by food contact with skin
Excluded: dermatitis caused by food eaten ( L27.2)
L24.7 Simple irritant contact dermatitis caused by plants other than food
L24.8 Simple irritant contact dermatitis caused by other substances. Dyes
L24.9 Simple irritant contact dermatitis, cause unspecified. Irritant contact eczema NOS

L25 Contact dermatitis, unspecified

Included: contact eczema, unspecified
Excludes: allergy NOS ( T78.4)
dermatitis:
NOS ( L30.9)
allergic contact ( L23. -)
caused by substances taken orally ( L27. -)
century ( H01.1)
simple irritable contact ( L24. -)
perioral ( L71.0)
eczema of the outer ear ( H60.5)
lesions of the skin and subcutaneous tissue associated
with exposure to radiation ( L55-L59)

L25.0 Unspecified contact dermatitis caused by cosmetics
L25.1 Unspecified contact dermatitis caused by drugs in contact with skin
If it is necessary to identify the drug, use an additional code for external causes (class XX).
Excludes: drug-induced allergy NOS ( T88.7)
drug-induced dermatitis ( L27.0-L27.1)
L25.2 Unspecified contact dermatitis caused by dyes
L25.3 Unspecified contact dermatitis caused by other chemicals. With cement. Insecticides
L25.4 Unspecified contact dermatitis caused by food in contact with skin
Excluded: contact dermatitis caused by food eaten ( L27.2)
L25.5 Unspecified contact dermatitis caused by plants other than food
L25.8 Unspecified contact dermatitis caused by other substances
L25.9 Unspecified contact dermatitis, cause not specified
Contact:
dermatitis (occupational) NOS
eczema (occupational) NOS

L26 Exfoliative dermatitis

Pitiriaz Gebra
Excludes: Ritter's disease ( L00)

L27 Dermatitis caused by ingested substances

Excluded: unfavorable:
drug exposure NOS ( T88.7)
reaction to food, excluding dermatitis ( T78.0-T78.1)
allergic reaction NOS ( T78.4)
contact dermatitis ( L23-l25)
medicinal:
photoallergic reaction ( L56.1)
phototoxic reaction ( L56.0)
urticaria ( L50. -)

L27.0 Generalized skin rash caused by drugs and medications
If it is necessary to identify the drug, use an additional code for external causes (class XX).
L27.1 Localized skin rash caused by drugs and medications
If it is necessary to identify the drug, use an additional code for external causes (class XX).
L27.2 Dermatitis caused by food eaten
Excluded: dermatitis caused by food in contact with skin ( L23.6, L24.6, L25.4)
L27.8 Dermatitis caused by other substances ingested
L27.9 Dermatitis due to unspecified substances ingested

L28 Simple chronic lichen and prurigo

L28.0 Simple chronic lichen. Limited neurodermatitis. Ringworm NOS
L28.1 Prurigo nodular
L28.2 Another pruritus
Pruritus:
NOS
Gebras
mitis
Urticaria papular

L29 Itching

Excluded: neurotic scratching of the skin ( L98.1)
psychogenic itching ( F45.8)

L29.0 Itching of the anus
L29.1 Itching of the scrotum
L29.2 Itching of the vulva
L29.3 Anogenital itching, unspecified
L29.8 Another itch
L29.9 Unspecified itching. Itching NOS

L30 Other dermatitis

Excluded: dermatitis:
contact ( L23-L25)
dry skin ( L85.3)
small plaque parapsoriasis ( L41.3)
stasis dermatitis ( I83.1-I83.2)

L30.0 Coin eczema
L30.1 Dyshidrosis [pompholyx]
L30.2 Skin autosensitization. Candida. Dermatophytosis. Eczematous
L30.3 Infectious dermatitis
Infectious eczema
L30.4 Erythematous diaper rash
L30.5 Pityriasis white
L30.8 Other specified dermatitis
L30.9 Dermatitis, unspecified
Eczema NOS

PAPULOSQUAMOUS DISORDERS (L40-L45)

L40 Psoriasis

L40.0 Psoriasis vulgaris. Coin psoriasis. Plaque
L40.1 Generalized pustular psoriasis. Impetigo herpetiformis. Zumbusch's disease
L40.2 Acrodermatitis persistent [Allopo]
L40.3 Palmar and plantar pustulosis
L40.4 Guttate psoriasis
L40.5+ Arthropathic psoriasis ( M07.0-M07.3*, M09.0*)
L40.8 Other psoriasis. Flexor inverse psoriasis
L40.9 Psoriasis, unspecified

L41 Parapsoriasis

Excludes: atrophic vascular poikiloderma ( L94.5)

L41.0 Pityriasis lichenoid and smallpox-like acute. Mucha-Habermann disease
L41.1 Pityriasis lichenoid chronic
L41.2 Lymphomatoid papulosis
L41.3 Small plaque parapsoriasis
L41.4 Large plaque parapsoriasis
L41.5 Reticular parapsoriasis
L41.8 Other parapsoriasis
L41.9 Parapsoriasis, unspecified

L42 Pityriasis rosea [Gibera]

L43 Lichen ruber flatus

Excluded: lichen planus pilaris ( L66.1)

L43.0 Lichen hypertrophic red flat
L43.1 Lichen planus bullosa
L43.2 Lichenoid reaction to a drug
If it is necessary to identify the drug, use an additional code for external causes (class XX).
L43.3 Lichen planus subacute (active). Tropical lichen planus
L43.8 Other lichen planus
L43.9 Lichen planus, unspecified

L44 Other papulosquamous changes

L44.0 Pityriasis red hair pityriasis
L44.1 Lichen brilliant
L44.2 Lichen linear
L44.3 Ringworm red moniliform
L44.4 Infantile papular acrodermatitis [Gianotti-Crosti syndrome]
L44.8 Other specified papulosquamous changes
L44.9 Papulosquamous changes, unspecified

L45* Papulosquamous disorders in diseases classified elsewhere

URTIA AND ERYTHEMA (L50-L54)

Excluded: Lyme disease ( A69.2)
rosacea ( L71. -)

L50 Urticaria

Excludes: allergic contact dermatitis ( L23. -)
angioedema ( T78.3)
hereditary vascular edema ( E88.0)
Quincke's edema ( T78.3)
hives:
giant ( T78.3)
newborn ( P83.8)
papular ( L28.2)
pigment ( Q82.2)
whey ( T80.6)
solar ( L56.3)

L50.0 Allergic urticaria
L50.1 Idiopathic urticaria
L50.2 Hives caused by exposure to low or high temperatures
L50.3 Dermatographic urticaria
L50.4 Vibration urticaria
L50.5 Cholinergic urticaria
L50.6 Contact urticaria
L50.8 Other hives
Hives:
chronic
periodic recurring
L50.9 Urticaria, unspecified

L51 Erythema multiforme

L51.0 Nonbullous erythema multiforme
L51.1 Bullous erythema multiforme. Stevens-Johnson syndrome
L51.2 Toxic epidermal necrolysis [Lyella]
L51.8 Other erythema multiforme
L51.9 Erythema multiforme, unspecified

L52 Erythema nodosum

L53 Other erythematous conditions

Excluded: erythema:
burn ( L59.0)
arising from contact with the skin of external agents ( L23-L25)
diaper rash ( L30.4)

L53.0 Erythema toxicum
If it is necessary to identify a toxic substance, use an additional external cause code (Class XX).
Excludes: neonatal erythema toxicum ( P83.1)
L53.1 Erythema annular centrifugal
L53.2 Erythema marginal
L53.3 Other chronic patterned erythema
L53.8 Other specified erythematous conditions
L53.9 Erythematous condition, unspecified. Erythema NOS. Erythroderma

L54* Erythema in diseases classified elsewhere

L54.0* Erythema marginal in acute articular rheumatism ( I00+)
L54.8* Erythema in other diseases classified elsewhere

DISEASES OF THE SKIN AND SUBcutaneous Fiber,
RADIATION EXPOSURE RELATED (L55-L59)

L55 Sunburn

L55.0 First degree sunburn
L55.1 Second degree sunburn
L55.2 Third degree sunburn
L55.8 Another sunburn
L55.9 Sunburn, unspecified

L56 Other acute skin changes caused by ultraviolet radiation

L56.0 Drug phototoxic reaction
If it is necessary to identify the drug, use an additional code for external causes (class XX).
L56.1 Drug photoallergic reaction
If it is necessary to identify the drug, use an additional code for external causes (class XX).
L56.2 Photocontact dermatitis
L56.3 Solar urticaria
L56.4 Polymorphic light eruption
L56.8 Other specified acute skin changes caused by ultraviolet radiation
L56.9 Acute skin change caused by ultraviolet radiation, unspecified

L57 Skin changes caused by chronic exposure to non-ionizing radiation

L57.0 Actinic (photochemical) keratosis
Keratosis:
NOS
senile
solar
L57.1 Actinic reticuloid
L57.2 Diamond-shaped skin on the back of the head (neck)
L57.3 Poikiloderma Siwatt
L57.4 Senile atrophy (flabbiness) of the skin. Senile elastosis
L57.5 Actinic [photochemical] granuloma
L57.8 Other skin changes caused by chronic exposure to non-ionizing radiation
Farmer's leather. Sailor's skin. Solar dermatitis
L57.9 Skin change caused by chronic exposure to non-ionizing radiation, unspecified

L58 Radiation radiation dermatitis

L58.0 Acute radiation dermatitis
L58.1 Chronic radiation dermatitis
L58.9 Radiation dermatitis, unspecified

L59 Other diseases of the skin and subcutaneous tissue associated with radiation

L59.0 Burn erythema [ab igne dermatitis]
L59.8 Other specified diseases of the skin and subcutaneous tissue associated with radiation
L59.9 Radiation-associated skin and subcutaneous tissue disease, unspecified

DISEASES OF SKIN APPENDIXES (L60-L75)

Excluded: congenital defects of the external integument ( Q84. -)

L60 Nail diseases

Excluded: clubbed nails ( R68.3)
onychia and paronychia ( L03.0)

L60.0 Ingrown nail
L60.1 Onycholysis
L60.2 Onychogryphosis
L60.3 Nail dystrophy
L60.4 Bo Lines
L60.5 Yellow nail syndrome
L60.8 Other nail diseases
L60.9 Nail disease, unspecified

L62* Changes in nails in diseases classified elsewhere

L62.0* Clubbed nail with pachydermoperiostosis ( M89.4+)
L62.8* Nail changes in other diseases classified elsewhere

L63 Alopecia areata

L63.0 Alopecia total
L63.1 Alopecia universalis
L63.2 Area baldness (ribbon form)
L63.8 Other alopecia areata
L63.9 Alopecia areata, unspecified

L64 Androgenetic alopecia

Included: male type baldness

L64.0 Drug-induced androgenetic alopecia
If it is necessary to identify the drug, use an additional code for external causes (class XX).
L64.8 Other androgenetic alopecia
L64.9 Androgenetic alopecia, unspecified

L65 Other non-scarring hair loss


Excluded: trichotillomania ( F63.3)

L65.0 Telogen hair loss
L65.1 Anagen hair loss. Regenerating Miasma
L65.2 Alopecia mucinosa
L65.8 Other specified non-scarring hair loss
L65.9 Non-scarring hair loss, unspecified

L66 Scarring alopecia

L66.0 Alopecia macular cicatricial
L66.1 Lichen planus pilaris. Follicular lichen planus
L66.2 Folliculitis leading to baldness
L66.3 Perifolliculitis of the head abscess
L66.4 Folliculitis reticularis cicatricial erythematous
L66.8 Other scarring alopecias
L66.9 Scarring alopecia, unspecified

L67 Abnormalities of hair and hair shaft color

Excluded: Knotty hair ( Q84.1)
beaded hair ( Q84.1)
telogen hair loss ( L65.0)

L67.0 Trichorrhexis nodosum
L67.1 Changes in hair color. Gray hair. Graying (premature). Hair heterochromia
Poliosis:
NOS
limited acquired
L67.8 Other abnormalities in hair color and hair shaft. Hair fragility
L67.9 Abnormality of hair and hair shaft color, unspecified

L68 Hypertrichosis

Included: excessive hairiness
Excluded: congenital hypertrichosis ( Q84.2)
resistant vellus hair ( Q84.2)

L68.0 Hirsutism
L68.1 Acquired vellus hair hypertrichosis
If it is necessary to identify the drug causing the disorder, use an additional external cause code (Class XX).
L68.2 Localized hypertrichosis
L68.3 Polytrichy
L68.8 Other hypertrichosis
L68.9 Hypertrichosis, unspecified

L70 Acne

Excluded: keloid acne ( L73.0)

L70.0 Common acne [acne vulgaris]
L70.1 Globular acne
L70.2 Acne pox. Necrotic miliary acne
L70.3 Tropical eels
L70.4 Baby acne
L70.5 Acne excoriee des jeunes filles
L70.8 Other acne
L70.9 Acne, unspecified

L71 Rosacea

L71.0 Perioral dermatitis
If it is necessary to identify the drug that caused the lesion, use an additional external cause code (class XX).
L71.1 Rhinophyma
L71.8 Another type of rosacea
L71.9 Rosacea unspecified

L72 Follicular cysts of the skin and subcutaneous tissue

L72.0 Epidermal cyst
L72.1 Trichodermal cyst. Hair cyst. Sebaceous cyst
L72.2 Styatocystoma multiple
L72.8 Other follicular cysts of the skin and subcutaneous tissue
L72.9 Follicular cyst of skin and subcutaneous tissue, unspecified

L73 Other diseases of hair follicles

L73.0 Acne keloids
L73.1 Pseudofolliculitis of beard hair
L73.2 Hidradenitis suppurativa
L73.8 Other specified follicular diseases. Sycosis of the beard
L73.9 Hair follicle disease, unspecified

L74 Diseases of merocrine [eccrine] sweat glands

Excluded: hyperhidrosis ( R61. -)

L74.0 Miliaria rubra
L74.1 Miliaria crystalline
L74.2 Prickly heat is deep. Tropical anhidrosis
L74.3 Miliaria unspecified
L74.4 Anhidrosis. Hypohidrosis
L74.8 Other diseases of merocrine sweat glands
L74.9 Merocrine sweating disorder, unspecified. Sweat gland damage NOS

L75 Diseases of apocrine sweat glands

Excludes: dyshidrosis [pompholyx] ( L30.1)
hidradenitis suppurativa ( L73.2)

L75.0 Bromhidrosis
L75.1 Chromhidrosis
L75.2 Apocrine heat rash. Fox-Fordyce disease
L75.8 Other diseases of the apocrine sweat glands
L75.9 Affection of apocrine sweat glands, unspecified

OTHER DISEASES OF THE SKIN AND SUBcutaneous Fiber (L80-L99)

L80 Vitiligo

L81 Other pigmentation disorders

Excludes: birthmark NOS ( Q82.5)
nevus - see Alphabetical index
Peutz-Jigers syndrome (Touraine) ( Q85.8)

L81.0 Post-inflammatory hyperpigmentation
L81.1 Chloasma
L81.2 Freckles
L81.3 Coffee stains
L81.4 Other melanin hyperpigmentation. Lentigo
L81.5 Leucoderma, not elsewhere classified
L81.6 Other disorders associated with decreased melanin production
L81.7 Pigmented red dermatosis. Angioma creeping
L81.8 Other specified pigmentation disorders. Iron pigmentation. Tattoo pigmentation
L81.9 Unspecified pigmentation disorder

L82 Seborrheic keratosis

Black papular dermatosis
Leser-Trélat disease

L83 Acanthosis nigricans

Confluent and reticulate papillomatosis

L84 Corns and calluses

Callus
Wedge-shaped callus (clavus)

L85 Other epidermal thickenings

Excluded: hypertrophic skin conditions ( L91. -)

L85.0 Acquired ichthyosis
Excluded: congenital ichthyosis ( Q80. -)
L85.1 Acquired keratosis [keratoderma] palmoplantar
Excluded: hereditary palmoplantar keratosis ( Q82.8)
L85.2 Keratosis punctate (palmar-plantar)
L85.3 Skin xerosis. Dry skin dermatitis
L85.8 Other specified epidermal thickenings. Cutaneous horn
L85.9 Epidermal thickening, unspecified

L86* Keratoderma in diseases classified elsewhere

Follicular keratosis) due to insufficiency
Xeroderma) vitamin A ( E50.8+)

L87 Transepidermal perforated changes

Excluded: granuloma annulare (perforated) ( L92.0)

L87.0 Keratosis follicular and parafollicular, penetrating the skin [Kierle disease]
Hyperkeratosis follicular penetrating
L87.1 Reactive perforating collagenosis
L87.2 Creeping perforating elastosis
L87.8 Other transepidermal perforation disorders
L87.9 Transepidermal perforation disorders, unspecified

L88 Pyoderma gangrenous

Gangrenous dermatitis
Necrotic pyoderma

L89 Decubital ulcer

Bedsore
Ulcer caused by plaster cast
Ulcer caused by compression
Excluded: decubital (trophic) cervical ulcer ( N86)

L90 Atrophic skin lesions

L90.0 Lichen sclerosis and atrophic
L90.1 Anetoderma Schwenninger-Buzzi
L90.2 Anetoderma Jadassohn-Pellisari
L90.3 Pasini-Pierini atrophoderma
L90.4 Acrodermatitis chronic atrophic
L90.5 Scar conditions and skin fibrosis. Soldered scar (skin). Scar. Disfigurement caused by a scar. Tripe NOS
Excluded: hypertrophic scar ( L91.0)
keloid scar ( L91.0)
L90.6 Atrophic stripes (striae)
L90.8 Other atrophic skin changes
L90.9 Atrophic skin change, unspecified

L91 Hypertrophic skin changes

L91.0 Keloid scar. Hypertrophic scar. Keloid
Excluded: acne keloids ( L73.0)
scar NOS ( L90.5)
L91.8 Other hypertrophic skin changes
L91.9 Hypertrophic skin change, unspecified

L92 Granulomatous changes in the skin and subcutaneous tissue

Excludes: actinic [photochemical] granuloma ( L57.5)

L92.0 Granuloma annular. Perforated granuloma annulare
L92.1 Necrobiosis lipoidica, not elsewhere classified
Excluded: associated with diabetes mellitus ( E10-E14)
L92.2 Facial granuloma [eosinophilic cutaneous granuloma]
L92.3 Granuloma of the skin and subcutaneous tissue caused by a foreign body
L92.8 Other granulomatous changes of the skin and subcutaneous tissue
L92.9 Granulomatous change of skin and subcutaneous tissue, unspecified

L93 Lupus erythematosus

Excluded: lupus:
ulcerative ( A18.4)
ordinary ( A18.4)
scleroderma ( M34. -)
systemic lupus erythematosus ( M32. -)
If it is necessary to identify the drug that caused the lesion, use an additional external cause code (class XX).
L93.0 Discoid lupus erythematosus. Lupus erythematosus NOS
L93.1 Subacute cutaneous lupus erythematosus
L93.2 Other limited lupus erythematosus. Lupus erythematosus deep. Lupus panniculitis

L94 Other localized connective tissue changes

Excluded: systemic connective tissue diseases ( M30-M36)

L94.0 Localized scleroderma. Limited scleroderma
L94.1 Linear scleroderma
L94.2 Calcification of the skin
L94.3 Sclerodactyly
L94.4 Gottron's papules
L94.5 Poikiloderma vascular atrophic
L94.6 Anyum [spontaneous dactylolysis]
L94.8 Other specified localized connective tissue changes
L94.9 Localized connective tissue change, unspecified

L95 Vasculitis limited to the skin, not elsewhere classified

Excluded: creeping angioma ( L81.7)
Henoch-Schönlein purpura ( D69.0)
hypersensitivity angiitis ( M31.0)
panniculitis:
NOS ( M79.3)
lupus ( L93.2)
neck and back ( M54.0)
recurrent (Weber-Christian) ( M35.6)
polyarteritis nodosa ( M30.0)
rheumatoid vasculitis ( M05.2)
serum sickness ( T80.6)
urticaria ( L50. -)
Wegener's granulomatosis ( M31.3)

L95.0 Vasculitis with marbled skin. White atrophy (plaque)
L95.1 Erythema sublime persistent
L95.8 Other vasculitides limited to the skin
L95.9 Vasculitis limited to skin, unspecified

L97 Ulcer of lower limb, not elsewhere classified

L89)
gangrene ( R02)
skin infections ( L00-L08)
A00-B99
varicose ulcer ( I83.0 , I83.2 )

L98 Other diseases of the skin and subcutaneous tissue, not elsewhere classified

L98.0 Pyogenic granuloma
L98.1 Artificial [artificial] dermatitis. Neurotic scratching of the skin
L98.2 Feverish neutrophilic dermatosis Sweet
L98.3 Wells' eosinophilic cellulitis
L98.4 Chronic skin ulcer, not elsewhere classified. Chronic skin ulcer NOS
Tropical ulcer NOS. Skin ulcer NOS
Excluded: decubital ulcer ( L89)
gangrene ( R02)
skin infections ( L00-L08)
specific infections classified under headings A00-B99
ulcer of the lower extremity NEC ( L97)
varicose ulcer ( I83.0 , I83.2 )
L98.5 Mucinosis of the skin. Focal mucinosis. Lichen myxedema
Excluded: focal mucinosis of the oral cavity ( K13.7)
myxedema ( E03.9)
L98.6 Other infiltrative diseases of the skin and subcutaneous tissue
Excluded: hyalinosis of the skin and mucous membranes ( E78.8)
L98.8 Other specified diseases of the skin and subcutaneous tissue
L98.9 Lesion of the skin and subcutaneous tissue, unspecified

L99* Other lesions of the skin and subcutaneous tissue in diseases classified elsewhere

L99.0* Cutaneous amyloidosis ( E85. -+)
Nodular amyloidosis. Patchy amyloidosis
L99.8* Other specified changes in the skin and subcutaneous tissue in diseases classified in other headings
Syphilitic:
alopecia ( A51.3+)
leucoderma ( A51.3+, A52.7+)

The disease is included in the International Classification of Diseases, 10th revision ICD-10. Due to the large number of etiological factors that can lead to the development of a necrotic process, according to the ICD, the code for a trophic ulcer can be found in different categories.

All variants of the ICD 10 code indicating a trophic ulcer on the leg

In the international classification, trophic ulcers of the lower extremities are classified as diseases of the skin and subcutaneous tissue. In this category, trophic ulcers belong to other diseases, i.e. not included in the rest of the classification. The subclass has nineteen sections listing various nutritional and skin pigmentation disorders that are not included in the classification in other subclasses.

The section that includes trophic ulcers is L98, diseases not included in other sections.

Subdivision – L98.4, chronic skin ulcer, not classified elsewhere. But this is a classification of an ulcer if its cause is not established.

Trophic ulcer with varicose veins has a completely different classification. Varicose veins belong to the class of diseases of the circulatory system, a subclass of diseases of the veins and lymphatic vessels.

For varicose veins of the lower extremities, a separate section I83 is allocated, which includes four different variants of the course of the disease, including I83.0 – varicose veins complicated by an ulcer, and I83.2 – varicose veins complicated by an ulcer and inflammation. Varicose veins with only inflammation, but without ulcers, are designated as I83.1, and uncomplicated varicose veins are designated I83.3.

Stages of development

  • Appearance
  • Cleansing
  • Granulation
  • Scarring

Initial stage The appearance of “lacquered” skin is characteristic. Redness and swelling appears. Liquid seeps through the “varnished” skin. Over time, the dead skin forms whitish spots, under which a scab forms. The first stage can last for several weeks.

At the second stage development of the ulcer has a bloody or mucopurulent discharge. If it has an unpleasant, pungent odor, this indicates the presence of an infection. During the cleansing stage, skin itching appears. As a rule, the second stage lasts about 1-1.5 months.

Healing process trophic wound depends on the quality of treatment. If you follow all the doctor’s recommendations, nutrition and tissue restoration in the area of ​​the ulcer are enhanced. Otherwise, a relapse occurs. Repeated trophic wounds are less susceptible to treatment. At the third stage, the wound surface begins to decrease.

Features of treatment

Trophic ulcers of the lower extremities are treated depending on the type of ulceration and the cause that provoked them. Treatment is prescribed on the basis of histological, bacteriological and cytological examination. Trophic ulcer of the leg can be treated in two ways:

  • Medication.
  • Surgical.